Ketogenic diet as primary AD therapy

Ketogenic diet as primary AD therapy enters the Adult Cognitive Disease corpus through the work of Masaki Hatakeyama (submission 47), and is indexed here as one of the concepts that submission puts to work.

The argument it belongs to

Masaki Hatakeyama's submission is summarised in this corpus as:

Alzheimer's disease is caused by intracellular essential amino acid deficiencies and decreased ATP synthesis efficiency resulting from excessive carbohydrate intake and persistent glycolytic pathway metabolism. Beta-amyloid and tau proteins are not causes but metabolites generated to remove excess non-essential amino acids from the imbalanced cytoplasm. A balanced ketogenic diet (40-50g carbohydrates/day) that promotes beta-oxidation of fat instead of glycolysis is the essential treatment.

Where it sits

The submission scores against the framework's convergence nodes as: compensatory paradigm 5 · neuroimmune interface 2.

Its declared subject matter: ketogenic diet, metabolism, amino acid deficiency, glycolysis, beta-oxidation, ATP synthesis, nutrition, carbohydrate excess.

Named by the same submission

3 other concepts enter the corpus through the same paper, so they cover adjacent ground: Glycolysis-fat oxidation metabolic switch · Metabolic origin of amyloid and tau · Nutritional rebalancing over pharmacological intervention.

Assembled from the corpus rather than written: the summary is quoted from the submission that named it; the node scores are read from its dossier. It has not yet been expanded into an article.